@gentleshaid first and foremost I appreciate the fact that you went through the write-up. Funnily enough, almost all your questions; the answers are already in the post. However, I'll try as much as possible to point you to them.

Your first question was,

I do not know why you referred to second trimester as the 6th month of pregnancy and the third trimester as 8-9 month of pregnancy.

Well, the truth is that I was trying to be specific in the actual months that these events occur that's why I included the months in brackets. Now, the point is even though the lanugo is believed to grow in the second trimester; it's specifically in the 6th month and not in the rest of the months that make up the second trimester. Same is applicable to the months that the shedding takes place.

Actually, I want to pinpoint the mistake in the next question before answering. You said,

...do you think these stimulators actually works knowing that hairs are dead cells?

In the article, I made this statement,

...know that hair growth begins inside the hair follicle and at that location the hair is regarded as a 'living' component.

So you see, the hair is not entirely a dead cell. As a matter of fact since the hair is made up of two structures like I rightly mentioned in the post, the part that are 'dead' are the shafts that are visible on the surfaces of the skin. In actuality, that's not where hair growth occurs.

Now, since we know that the hair follicles are living cells, you'll agree with me that hair growth stimulators do work. Although I can't exactly conclude the mechanism behind this, but I feel the answer might just be what I already said in the post.

According to findings done by Dr. Sarah E. Millar in her article on 'Why some parts of the body have hair and others don't', she stated that the answer is embedded in the fact that there is presence of a naturally-occurring inhibitor called Dickkopf 2 (DKK2) in these hairless regions. Now, the action of this inhibitor is exerted on WNT signaling pathway responsible for controlling hair growth. That's why the hair follicles are entirely turned off in these regions.

The hair growth stimulator may interfere with Dickkopf 2 (DKK2) and in the process hair follicles are automatically turned on thereby resulting in hair growth.

Concerning this particular question,

I have seen a lot of females having beards; is this a genetic condition or a kind of environmentally acquired trait?

Here's the answer already in the post...

As a matter of fact, the females you see having facial hairs (beards), hairs on the chest, etc., might either be suffering from androgen hormonal imbalance (caused by increased level of androgen) or a rare genetic disorder.

For the last question, honestly I just got to know about it from you. 😂

Thanks for stopping by. I really appreciate.